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Peptide Comparisons

Comprehensive side-by-side comparisons of research peptides and substances. Understand the differences, similarities, and optimal use cases for informed research decisions.

Ozempic/Wegovy (Semaglutide) vs Retatrutide

Ozempic/Wegovy (Semaglutide)

GLP-1 Receptor Agonist

Description

Semaglutide is marketed as Ozempic (for type 2 diabetes) and Wegovy (for weight management). It's a selective GLP-1 receptor agonist that has become one of the most popular weight loss medications globally due to proven efficacy and safety.

Mechanism of Action

Selective GLP-1 receptor agonist that mimics the incretin hormone glucagon-like peptide-1. Enhances glucose-dependent insulin secretion, suppresses glucagon release, slows gastric emptying significantly, and reduces appetite through central nervous system effects on satiety centers in the hypothalamus.

Key Benefits

  • 15-17% average body weight reduction (STEP trials)
  • Improved glycemic control and HbA1c reduction
  • Reduced cardiovascular events by 20% (SELECT trial)
  • Decreased appetite and food cravings
  • FDA-approved with extensive real-world safety data
  • Well-tolerated in most subjects
  • Convenient once-weekly dosing
  • Improved blood pressure and lipid profiles
Dosing: Weekly subcutaneous injection. Ozempic: 0.25mg start, titrate to 0.5-2mg. Wegovy: 0.25mg start, titrate to 2.4mg over 16-20 weeks.
Duration: Half-life of approximately 7 days, enabling reliable once-weekly administration
VS

Retatrutide

Triple GIP/GLP-1/Glucagon Receptor Agonist

Description

Next-generation triple agonist currently in Phase 3 clinical trials. Retatrutide adds glucagon receptor activation to GLP-1 and GIP mechanisms, creating the most comprehensive metabolic intervention available in research.

Mechanism of Action

Triple-action mechanism: (1) GLP-1 activation for appetite suppression and glucose control, (2) GIP activation for enhanced insulin sensitivity and fat metabolism, (3) Glucagon activation for increased energy expenditure and fat oxidation. The glucagon component is revolutionary—it boosts metabolic rate while the other pathways control appetite.

Key Benefits

  • 24-28% average body weight reduction (Phase 2 trials) - HIGHEST of any agent
  • Increased energy expenditure through glucagon-mediated thermogenesis
  • Enhanced fat oxidation beyond simple appetite suppression
  • Better preservation of lean muscle mass during weight loss
  • Improved metabolic flexibility and insulin sensitivity
  • Faster weight loss compared to single or dual agonists
  • Potential for superior body composition changes
Dosing: Weekly subcutaneous injection in research settings. Phase 3 trials using 4-12mg doses.
Duration: Extended half-life of 5-7 days, similar to other long-acting metabolic peptides

The Verdict

Semaglutide (Ozempic/Wegovy) delivers proven 15-17% weight loss with FDA approval and years of safety data—it's the gold standard for clinical use today. Retatrutide shows superior 24-28% weight loss (40-60% more than semaglutide) through its unique triple-agonist mechanism, particularly glucagon activation which increases metabolic rate. However, Retatrutide is investigational only. For current treatment, semaglutide is the clear choice. For cutting-edge research, Retatrutide represents the future.

Key Considerations

  • Semaglutide: 15-17% weight loss | Retatrutide: 24-28% weight loss
  • Semaglutide is FDA-approved and available; Retatrutide is research-only
  • Retatrutide's glucagon component increases energy expenditure (unique advantage)
  • Both require gradual dose titration to minimize GI side effects
  • Semaglutide has cardiovascular outcome data; Retatrutide trials ongoing
  • Cost and availability strongly favor semaglutide currently
  • Retatrutide may better preserve muscle mass during weight loss

Mounjaro/Zepbound (Tirzepatide) vs Retatrutide

Mounjaro/Zepbound (Tirzepatide)

Dual GIP/GLP-1 Receptor Agonist

Description

Tirzepatide is marketed as Mounjaro (for type 2 diabetes) and Zepbound (for weight management). It's the first dual GIP/GLP-1 agonist, representing a significant advancement over single GLP-1 agonists like semaglutide.

Mechanism of Action

Dual-action mechanism activating both GIP (glucose-dependent insulinotropic polypeptide) and GLP-1 (glucagon-like peptide-1) receptors. GLP-1 provides appetite suppression and glucose control, while GIP enhances insulin sensitivity, promotes fat metabolism, and provides additional metabolic benefits. The combination produces superior weight loss compared to GLP-1 alone.

Key Benefits

  • 20-22% average body weight reduction (SURMOUNT trials) - superior to semaglutide
  • Superior glycemic control compared to GLP-1-only agonists
  • Reduced cardiovascular risk markers
  • Enhanced insulin sensitivity through dual-pathway activation
  • Decreased appetite and improved satiety
  • FDA-approved with growing real-world evidence
  • Better tolerability profile than some GLP-1 agonists
Dosing: Weekly subcutaneous injection. Start at 2.5mg, titrate to 5-15mg based on tolerance and response over 20+ weeks.
Duration: Half-life of approximately 5 days, allowing reliable once-weekly dosing
VS

Retatrutide

Triple GIP/GLP-1/Glucagon Receptor Agonist

Description

Investigational triple agonist that takes tirzepatide's dual mechanism and adds glucagon receptor activation. This creates the most comprehensive metabolic intervention by addressing appetite, insulin sensitivity, AND energy expenditure simultaneously.

Mechanism of Action

Triple-action: (1) GLP-1 for appetite/glucose control, (2) GIP for insulin sensitivity/fat metabolism, (3) Glucagon for increased energy expenditure and fat oxidation. The glucagon component is the key differentiator—it actively increases metabolic rate and thermogenesis, creating a dual approach: reduce calories in (GLP-1) AND increase calories out (glucagon).

Key Benefits

  • 24-28% average body weight reduction - 20-35% more than tirzepatide
  • Increased basal metabolic rate through glucagon activation
  • Enhanced lipolysis and fat oxidation
  • Superior lean mass preservation during weight loss
  • Improved metabolic flexibility
  • Faster rate of weight loss compared to dual agonists
  • Potential for better long-term weight maintenance
Dosing: Weekly subcutaneous injection in research settings, 4-12mg range in clinical trials.
Duration: Extended half-life of 5-7 days

The Verdict

Tirzepatide (Mounjaro/Zepbound) delivers impressive 20-22% weight loss through dual GIP/GLP-1 activation—currently the most effective FDA-approved weight loss medication. Retatrutide shows even greater 24-28% weight loss by adding glucagon receptor activation, which uniquely increases energy expenditure. The difference: tirzepatide controls appetite and improves metabolism; Retatrutide does both PLUS actively increases calorie burning. However, tirzepatide is FDA-approved and available now, while Retatrutide is investigational.

Key Considerations

  • Tirzepatide: 20-22% weight loss | Retatrutide: 24-28% weight loss
  • Tirzepatide is FDA-approved; Retatrutide is investigational
  • Retatrutide's glucagon component increases metabolic rate (unique advantage)
  • Both require careful dose titration over several months
  • Tirzepatide has established safety profile; Retatrutide in Phase 3 trials
  • GI side effects similar between both (nausea, vomiting during titration)
  • Retatrutide may have different side effect profile due to glucagon activation

Saxenda/Victoza (Liraglutide) vs Retatrutide

Saxenda/Victoza (Liraglutide)

GLP-1 Receptor Agonist (First Generation)

Description

Liraglutide, marketed as Victoza (diabetes) and Saxenda (weight loss), was one of the first long-acting GLP-1 agonists. While now superseded by newer agents like semaglutide and tirzepatide, it remains a proven and accessible option.

Mechanism of Action

GLP-1 receptor agonist that enhances insulin secretion, suppresses glucagon, slows gastric emptying, and reduces appetite. Shorter half-life than newer GLP-1 agonists, requiring daily administration. Works through the same pathways as semaglutide but with less potency.

Key Benefits

  • 5-10% average body weight reduction - modest compared to newer agents
  • Improved glycemic control in diabetic subjects
  • Cardiovascular benefits demonstrated in LEADER trial
  • Reduced appetite and caloric intake
  • Long track record of safety and tolerability
  • Lower cost than newer GLP-1 agonists in some markets
  • Well-established dosing protocols
Dosing: Daily subcutaneous injection. Saxenda: start 0.6mg, titrate to 3mg over 5 weeks. Victoza: 0.6mg start, up to 1.8mg.
Duration: Shorter half-life of ~13 hours, requiring daily dosing (less convenient than weekly options)
VS

Retatrutide

Triple GIP/GLP-1/Glucagon Receptor Agonist

Description

Third-generation triple agonist representing a quantum leap beyond first-generation GLP-1 agonists like liraglutide. Adds GIP and glucagon pathways to create comprehensive metabolic intervention.

Mechanism of Action

Triple-action mechanism: GLP-1 (appetite/glucose), GIP (insulin sensitivity/metabolism), Glucagon (energy expenditure/fat oxidation). This multi-pathway approach addresses weight loss through multiple complementary mechanisms rather than GLP-1 alone.

Key Benefits

  • 24-28% average body weight reduction - 3-5X more than liraglutide
  • Increased energy expenditure (not possible with GLP-1 alone)
  • Enhanced fat oxidation and lipolysis
  • Better lean mass preservation
  • Superior metabolic improvements
  • Once-weekly dosing vs daily for liraglutide
  • Represents cutting-edge metabolic science
Dosing: Weekly subcutaneous injection, 4-12mg in research settings.
Duration: Extended 5-7 day half-life, enabling weekly administration

The Verdict

Liraglutide (Saxenda/Victoza) delivers modest 5-10% weight loss and requires daily injections—it's a proven first-generation option but clearly outperformed by newer agents. Retatrutide achieves 24-28% weight loss (3-5X more) with weekly dosing through its revolutionary triple-agonist mechanism. The difference is dramatic: liraglutide is single-pathway GLP-1 only; Retatrutide is multi-pathway with metabolic rate enhancement. Liraglutide is FDA-approved and available; Retatrutide is investigational but represents the future.

Key Considerations

  • Liraglutide: 5-10% weight loss | Retatrutide: 24-28% weight loss
  • Liraglutide requires daily injections; Retatrutide is once-weekly
  • Retatrutide is 3-5X more effective for weight loss
  • Liraglutide is FDA-approved and widely available
  • Cost: Liraglutide may be more affordable in some markets
  • Liraglutide has longest safety track record among GLP-1 agonists
  • Retatrutide represents next-generation technology vs first-generation liraglutide

Trulicity (Dulaglutide) vs Retatrutide

Trulicity (Dulaglutide)

GLP-1 Receptor Agonist

Description

Dulaglutide (Trulicity) is a once-weekly GLP-1 agonist primarily used for type 2 diabetes, with modest weight loss effects. It features a unique single-dose pen delivery system for convenience.

Mechanism of Action

GLP-1 receptor agonist that enhances glucose-dependent insulin secretion, suppresses glucagon, slows gastric emptying, and reduces appetite. Similar mechanism to other GLP-1 agonists but with moderate potency between liraglutide and semaglutide.

Key Benefits

  • 5-10% average body weight reduction - modest weight loss effect
  • Improved glycemic control in diabetic subjects
  • Cardiovascular benefits demonstrated in REWIND trial
  • Convenient once-weekly dosing
  • User-friendly single-dose pen device
  • Well-tolerated with established safety profile
  • Reduced risk of hypoglycemia
Dosing: Weekly subcutaneous injection via single-dose pen. Start 0.75mg, can increase to 1.5mg or 3mg (diabetes) or 4.5mg (weight loss).
Duration: Half-life of approximately 5 days, enabling once-weekly administration
VS

Retatrutide

Triple GIP/GLP-1/Glucagon Receptor Agonist

Description

Next-generation triple agonist that goes far beyond single-pathway GLP-1 agonists like dulaglutide. Combines three complementary mechanisms for superior metabolic effects.

Mechanism of Action

Triple-action: GLP-1 (appetite suppression/glucose control), GIP (insulin sensitivity/fat metabolism), Glucagon (energy expenditure/thermogenesis). The multi-pathway approach creates synergistic effects impossible with GLP-1 alone.

Key Benefits

  • 24-28% average body weight reduction - 3-5X more than dulaglutide
  • Increased basal metabolic rate through glucagon activation
  • Enhanced fat oxidation and lipolysis
  • Superior lean muscle mass preservation
  • Improved metabolic flexibility
  • Comprehensive metabolic intervention
  • Represents cutting-edge peptide research
Dosing: Weekly subcutaneous injection in research settings, 4-12mg doses in trials.
Duration: Extended half-life of 5-7 days

The Verdict

Dulaglutide (Trulicity) provides 5-10% weight loss with convenient weekly dosing and proven cardiovascular benefits—it's a solid FDA-approved option for diabetes with modest weight loss. Retatrutide delivers 24-28% weight loss (3-5X more) through its revolutionary triple-agonist mechanism that includes metabolic rate enhancement. The difference is substantial: dulaglutide is single-pathway; Retatrutide is multi-pathway with energy expenditure boost. Dulaglutide is available now; Retatrutide is investigational.

Key Considerations

  • Dulaglutide: 5-10% weight loss | Retatrutide: 24-28% weight loss
  • Both offer convenient once-weekly dosing
  • Dulaglutide is FDA-approved; Retatrutide is investigational
  • Retatrutide is 3-5X more effective for weight loss
  • Dulaglutide has established cardiovascular outcome data
  • Dulaglutide's pen device is very user-friendly
  • Retatrutide represents next-generation vs current-generation technology

Semax vs SSRIs (Prozac, Zoloft, Lexapro)

Semax

Synthetic ACTH Analog (Nootropic Peptide)

Description

Russian-developed synthetic peptide derived from ACTH with powerful nootropic and mood-enhancing properties. Unlike traditional antidepressants, Semax enhances cognitive function while improving mood through neuroplasticity mechanisms.

Mechanism of Action

Increases BDNF (brain-derived neurotrophic factor) expression, enhances neuroplasticity, modulates dopamine and serotonin systems without direct receptor binding. Improves cerebral blood flow and oxygen utilization. Provides neuroprotection and promotes neurogenesis. Works through growth factor pathways rather than neurotransmitter reuptake inhibition.

Key Benefits

  • Improved mood and emotional stability
  • Enhanced cognitive function and mental clarity (unlike SSRIs)
  • Increased focus, concentration, and mental energy
  • Improved stress resilience
  • No sexual dysfunction or weight gain
  • No emotional blunting or numbness
  • Rapid onset (days vs weeks for SSRIs)
  • No withdrawal syndrome
  • Neuroprotective and neurogenic effects
Dosing: Intranasal administration: 2-3 drops (400-600mcg) per nostril, 1-2 times daily. Higher concentrations (0.1%, 1%) available.
Duration: Short half-life but neuroplastic effects accumulate with consistent use
VS

SSRIs (Prozac, Zoloft, Lexapro)

Selective Serotonin Reuptake Inhibitors

Description

Standard pharmaceutical antidepressants that work by increasing serotonin levels in the brain. SSRIs include fluoxetine (Prozac), sertraline (Zoloft), escitalopram (Lexapro), and others. They're first-line treatment for depression and anxiety disorders.

Mechanism of Action

Block the reuptake of serotonin at the presynaptic neuron, increasing serotonin availability in the synaptic cleft. This enhances serotonergic neurotransmission, which over weeks leads to downstream changes in receptor sensitivity and neural circuit function that improve mood.

Key Benefits

  • Proven efficacy for major depressive disorder
  • Effective for anxiety disorders
  • FDA-approved with decades of safety data
  • Well-established dosing protocols
  • Reduces suicidal ideation in depression
  • Helpful for OCD and panic disorder
  • Generally well-tolerated long-term
Dosing: Oral daily administration. Prozac: 20-80mg. Zoloft: 50-200mg. Lexapro: 10-20mg. Requires 4-6 weeks for full effect.
Duration: Long half-lives (24-96 hours depending on agent), requiring daily dosing

The Verdict

SSRIs are proven, FDA-approved treatments for depression and anxiety with decades of evidence—they're the standard of care for moderate to severe depression. However, they cause sexual dysfunction (60-70% of users), weight gain, emotional blunting, and take 4-6 weeks to work. Semax offers rapid mood improvement (days), enhances cognition rather than impairing it, has no sexual or weight side effects, and works through neuroplasticity rather than neurotransmitter manipulation. For severe clinical depression, SSRIs remain first-line. For mood optimization, cognitive enhancement, and stress resilience without side effects, Semax is superior.

Key Considerations

  • SSRIs cause sexual dysfunction in 60-70% of users; Semax does not
  • SSRIs often cause weight gain; Semax does not
  • SSRIs cause emotional blunting; Semax enhances emotional clarity
  • SSRIs impair cognition in some users; Semax enhances cognition
  • SSRIs take 4-6 weeks to work; Semax works within days
  • SSRIs have withdrawal syndrome; Semax does not
  • SSRIs are FDA-approved for depression; Semax is research-only
  • SSRIs are better for severe clinical depression; Semax for optimization

Selank vs Benzodiazepines (Xanax, Valium, Ativan)

Selank

Synthetic Anxiolytic Peptide (Tuftsin Analog)

Description

Russian-developed synthetic peptide with powerful anxiolytic effects without sedation, dependence, or cognitive impairment. Represents a revolutionary approach to anxiety management that enhances rather than impairs function.

Mechanism of Action

Modulates GABA, serotonin, and dopamine systems without direct receptor binding. Increases BDNF, enhances enkephalin metabolism, and stabilizes emotional regulation through multiple neurotransmitter pathways. Provides anxiolytic effects while maintaining or enhancing cognitive function. No CNS depression.

Key Benefits

  • Powerful anxiolytic effects without sedation
  • Enhanced cognitive function and mental clarity
  • Improved focus and concentration under stress
  • Better stress resilience and emotional stability
  • No tolerance or dependence development
  • No withdrawal syndrome
  • No cognitive or motor impairment
  • No addiction potential
  • Immune system modulation
  • Improved sleep quality without sedation
Dosing: Intranasal: 2-3 drops (400-600mcg) per nostril, 2-3 times daily. Can be used as needed or daily.
Duration: Short half-life but effects on neurotransmitter systems persist throughout the day
VS

Benzodiazepines (Xanax, Valium, Ativan)

GABA-A Receptor Positive Allosteric Modulators

Description

Pharmaceutical anxiolytics including alprazolam (Xanax), diazepam (Valium), lorazepam (Ativan), and others. Benzodiazepines are highly effective for acute anxiety but carry significant risks of dependence, tolerance, and cognitive impairment.

Mechanism of Action

Enhance GABA-A receptor function by increasing the frequency of chloride channel opening. This produces rapid anxiolytic, sedative, muscle relaxant, and anticonvulsant effects through CNS depression. The mechanism provides powerful acute relief but causes tolerance and dependence with regular use.

Key Benefits

  • Rapid, powerful anxiolytic effects (15-30 minutes)
  • Effective for panic attacks and acute severe anxiety
  • Muscle relaxation properties
  • Anticonvulsant effects
  • Well-established clinical use
  • Effective for acute situational anxiety
  • Immediate relief in crisis situations
Dosing: Oral administration. Xanax: 0.25-2mg 2-4x daily. Valium: 2-10mg 2-4x daily. Ativan: 1-2mg 2-3x daily.
Duration: Variable half-lives: Xanax (short, 11hrs), Ativan (intermediate, 12hrs), Valium (long, 20-100hrs)

The Verdict

Benzodiazepines provide rapid, powerful anxiety relief and are essential for acute severe anxiety, panic attacks, or crisis situations. However, they cause sedation, cognitive impairment, tolerance, physical dependence, and dangerous withdrawal. Selank offers comparable anxiolytic effects WITHOUT sedation, impairment, dependence, or withdrawal—while actually enhancing cognitive function. For acute severe anxiety or panic, benzodiazepines are more powerful. For daily anxiety management, stress resilience, and performance optimization, Selank is vastly superior with no addiction risk.

Key Considerations

  • Benzodiazepines cause dependence and tolerance; Selank does not
  • Benzodiazepines impair cognition and motor function; Selank enhances cognition
  • Benzodiazepines cause sedation; Selank does not
  • Benzodiazepines have dangerous withdrawal; Selank has none
  • Benzodiazepines are controlled substances; Selank is unscheduled
  • Benzodiazepines are more powerful for acute severe anxiety
  • Selank is superior for long-term daily anxiety management
  • Benzodiazepines are FDA-approved; Selank is research-only

Human Growth Hormone (HGH) vs Muscle Stack (CJC-1295 + Ipamorelin)

Human Growth Hormone (HGH)

Recombinant Growth Hormone

Description

Synthetic version of naturally occurring human growth hormone (somatropin). HGH is a 191-amino acid peptide hormone that directly replaces endogenous growth hormone.

Mechanism of Action

Direct exogenous administration of growth hormone, bypassing the body's natural production mechanisms. HGH binds to growth hormone receptors throughout the body, stimulating IGF-1 production in the liver and promoting anabolic effects in muscle, bone, and connective tissue.

Key Benefits

  • Rapid and pronounced increases in IGF-1 levels
  • Significant lean muscle mass gains
  • Enhanced fat loss, particularly visceral fat
  • Improved recovery and tissue repair
  • Increased bone density
  • Better skin quality and collagen synthesis
  • Predictable dosing and response
Dosing: Daily subcutaneous injections, typically 2-4 IU for anti-aging, 4-8 IU for body composition, higher doses for athletic purposes
Duration: Short half-life of 2-3 hours, requiring daily administration
VS

Muscle Stack (CJC-1295 + Ipamorelin)

Growth Hormone Secretagogue Combination

Description

Synergistic combination of CJC-1295 (GHRH analog) and Ipamorelin (GHRP). This stack stimulates the body's natural growth hormone production through two complementary pathways.

Mechanism of Action

Dual-pathway stimulation: CJC-1295 amplifies natural GH pulses by mimicking growth hormone-releasing hormone, while Ipamorelin triggers GH release through ghrelin receptor activation. Together they create larger, more frequent GH pulses while maintaining natural pulsatile patterns.

Key Benefits

  • Maintains natural pulsatile GH release patterns
  • No suppression of endogenous GH production
  • Increased lean muscle mass (more gradual than HGH)
  • Enhanced fat metabolism
  • Improved sleep quality and recovery
  • Better safety profile with fewer side effects
  • No impact on cortisol or prolactin (Ipamorelin selectivity)
  • More affordable than pharmaceutical HGH
Dosing: Typically administered together: CJC-1295 (100-200mcg) + Ipamorelin (100-300mcg), 1-2 times daily, often before bed and post-workout
Duration: CJC-1295 NO DAC: 6-8 hour half-life; Ipamorelin: 2 hour half-life

The Verdict

HGH provides more dramatic and immediate results with higher IGF-1 elevations, making it superior for rapid body composition changes. However, the Muscle Stack (CJC-1295 + Ipamorelin) offers a more physiological approach that maintains natural GH patterns, doesn't suppress endogenous production, has fewer side effects, and costs significantly less. For long-term use and overall health optimization, the peptide stack is often preferred; for maximum anabolic effects, HGH is more powerful.

Key Considerations

  • HGH can suppress natural GH production; peptide stack does not
  • HGH carries higher risk of side effects (edema, carpal tunnel, insulin resistance)
  • Peptide stack is significantly more affordable
  • HGH provides faster, more dramatic results
  • Peptide stack maintains healthier pulsatile GH patterns
  • HGH requires daily injections; peptide stack offers flexible dosing
  • Legal and regulatory considerations differ significantly

Diazepam vs Selank

Diazepam

Benzodiazepine (GABA-A Receptor Modulator)

Description

Classic pharmaceutical anxiolytic medication. Diazepam is a long-acting benzodiazepine that has been used for decades to treat anxiety, muscle spasms, and seizures.

Mechanism of Action

Enhances the effect of GABA (gamma-aminobutyric acid) at GABA-A receptors by increasing the frequency of chloride channel opening. This produces rapid anxiolytic, sedative, muscle relaxant, and anticonvulsant effects through CNS depression.

Key Benefits

  • Rapid onset of anxiolytic effects (30-60 minutes)
  • Powerful acute anxiety relief
  • Muscle relaxation properties
  • Anticonvulsant effects
  • Long duration of action (20-100 hour half-life)
  • Well-established clinical use and dosing
  • Effective for panic attacks and acute anxiety
Dosing: Oral administration typically 2-10mg, 2-4 times daily depending on indication. Injectable forms available for acute situations.
Duration: Very long half-life (20-100 hours including active metabolites), providing extended anxiolytic effects
VS

Selank

Synthetic Anxiolytic Peptide (Tuftsin Analog)

Description

Russian-developed synthetic peptide derived from the immunomodulatory peptide tuftsin. Selank is a heptapeptide with anxiolytic and nootropic properties without sedation or dependence.

Mechanism of Action

Modulates GABA, serotonin, and dopamine systems without direct receptor binding. Increases BDNF (brain-derived neurotrophic factor), enhances enkephalin metabolism, and stabilizes emotional regulation through multiple neurotransmitter pathways. Provides anxiolytic effects while maintaining or enhancing cognitive function.

Key Benefits

  • Anxiolytic effects without sedation
  • Enhanced cognitive function and mental clarity
  • Improved focus and concentration
  • Stress resilience and emotional stability
  • No tolerance or dependence development
  • No withdrawal symptoms
  • Neuroprotective properties
  • Immune system modulation
  • No impairment of motor function or memory
Dosing: Intranasal administration: 2-3 drops (400-600mcg) per nostril, 2-3 times daily. Subcutaneous injection: 250-500mcg daily in research settings.
Duration: Short half-life (minutes to hours), but effects on neurotransmitter systems persist longer

The Verdict

Diazepam provides more powerful acute anxiety relief and is essential for severe anxiety, panic attacks, or medical situations requiring rapid intervention. However, Selank offers a superior profile for chronic anxiety management: no sedation, no cognitive impairment, no dependence risk, and additional cognitive enhancement. Selank is ideal for daily stress management and performance optimization, while diazepam is better suited for acute, severe anxiety or short-term use.

Key Considerations

  • Diazepam carries significant risk of dependence and tolerance
  • Selank has no addiction potential or withdrawal
  • Diazepam causes sedation and cognitive impairment; Selank enhances cognition
  • Diazepam is more powerful for acute severe anxiety
  • Selank is superior for long-term daily use
  • Diazepam has drug interactions and contraindications; Selank has minimal
  • Legal status differs: Diazepam is controlled; Selank is unscheduled in most regions

BPC-157 vs TB-500

BPC-157

Body Protection Compound (Gastric Peptide Derivative)

Description

Synthetic peptide derived from a protective protein found in gastric juice. BPC-157 has remarkable healing properties across multiple tissue types with particular efficacy for gut health and tendon/ligament repair.

Mechanism of Action

Promotes angiogenesis through VEGF (vascular endothelial growth factor) upregulation, enhances fibroblast activity, modulates growth factor expression, and stabilizes cellular junctions. Particularly effective at healing gastrointestinal tissue, tendons, ligaments, and muscle. Also exhibits anti-inflammatory and neuroprotective effects.

Key Benefits

  • Accelerated tendon and ligament healing
  • Superior gut healing and GI tract repair
  • Reduced inflammation systemically
  • Enhanced wound healing
  • Neuroprotective effects
  • Improved joint health
  • Faster recovery from injuries
  • May protect against NSAID-induced damage
Dosing: Subcutaneous or intramuscular injection: 250-500mcg once or twice daily, often injected near injury site for localized effects. Oral administration also researched.
Duration: Short half-life but tissue-level effects persist for extended periods
VS

TB-500

Thymosin Beta-4 Fragment

Description

Synthetic version of Thymosin Beta-4, a naturally occurring peptide present in high concentrations in blood platelets, wound fluid, and other tissues. TB-500 is the active fragment responsible for healing and regeneration.

Mechanism of Action

Promotes cell migration, proliferation, and differentiation through actin regulation. Upregulates actin polymerization, enabling cells to migrate to injury sites. Stimulates angiogenesis, reduces inflammation, and promotes tissue remodeling. Particularly effective for systemic healing and flexibility enhancement.

Key Benefits

  • Systemic tissue repair and regeneration
  • Enhanced flexibility and range of motion
  • Reduced acute and chronic inflammation
  • Accelerated wound healing
  • Improved hair growth (research finding)
  • Cardiovascular protective effects
  • Reduced fibrous tissue formation
  • Faster recovery from muscle strains
Dosing: Subcutaneous injection: Loading phase of 5-10mg twice weekly for 4-6 weeks, then maintenance of 2-5mg weekly. Systemic effects allow injection anywhere.
Duration: Long half-life and tissue retention, allowing less frequent dosing than BPC-157

The Verdict

BPC-157 excels at localized healing, particularly for tendons, ligaments, and gut issues, with faster onset of effects. TB-500 provides superior systemic healing, better flexibility improvements, and longer-lasting effects with less frequent dosing. Many researchers use both together (the "Wolverine Stack") for synergistic healing through complementary mechanisms.

Key Considerations

  • BPC-157 is better for gut health and localized tendon/ligament injuries
  • TB-500 is superior for systemic healing and flexibility
  • BPC-157 requires more frequent dosing but has faster onset
  • TB-500 has longer-lasting effects with less frequent administration
  • Combining both provides synergistic healing benefits
  • BPC-157 can be taken orally for GI issues; TB-500 requires injection
  • Both have excellent safety profiles with minimal side effects

Semaglutide vs Retatrutide

Semaglutide

GLP-1 Receptor Agonist

Description

FDA-approved GLP-1 receptor agonist marketed as Ozempic (diabetes) and Wegovy (weight loss). Semaglutide has become one of the most popular weight loss medications due to its efficacy and safety profile.

Mechanism of Action

Selective GLP-1 receptor agonist that mimics the incretin hormone GLP-1. Enhances insulin secretion, suppresses glucagon, slows gastric emptying, and reduces appetite through central nervous system effects. The single-pathway mechanism provides reliable, predictable effects.

Key Benefits

  • Significant weight loss (15-17% body weight reduction)
  • Improved glycemic control
  • Reduced cardiovascular events in high-risk subjects
  • Decreased appetite and food cravings
  • FDA-approved with extensive safety data
  • Well-tolerated in most subjects
  • Convenient once-weekly dosing
Dosing: Weekly subcutaneous injection, starting at 0.25mg and titrating up to 1-2.4mg over 16-20 weeks
Duration: Half-life of approximately 7 days, enabling once-weekly administration
VS

Retatrutide

Triple GIP/GLP-1/Glucagon Receptor Agonist

Description

Investigational triple agonist representing next-generation metabolic therapy. Retatrutide adds glucagon receptor activation to the GLP-1 mechanism, creating a more comprehensive metabolic intervention.

Mechanism of Action

Triple-action mechanism: GLP-1 for appetite suppression and glucose control, GIP for enhanced insulin sensitivity and metabolic benefits, and glucagon for increased energy expenditure and fat oxidation. The glucagon component is key to superior weight loss by boosting metabolic rate.

Key Benefits

  • Superior weight loss (24-28% body weight reduction)
  • Increased energy expenditure
  • Enhanced fat oxidation beyond appetite suppression
  • Better preservation of lean muscle mass
  • Improved metabolic flexibility
  • Potentially faster results than single or dual agonists
Dosing: Weekly subcutaneous injection in research settings, doses of 4-12mg in clinical trials
Duration: Extended half-life of 5-7 days, similar to other long-acting metabolic peptides

The Verdict

Semaglutide is the proven, FDA-approved option with excellent efficacy (15-17% weight loss), established safety, and real-world availability. Retatrutide shows superior weight loss (24-28%) in clinical trials through its unique triple-agonist mechanism, particularly the addition of glucagon receptor activation which increases energy expenditure. However, Retatrutide is still investigational. For current clinical use, Semaglutide is the clear choice; for cutting-edge research, Retatrutide represents the future of metabolic therapy.

Key Considerations

  • Semaglutide is FDA-approved; Retatrutide is investigational only
  • Retatrutide shows 40-60% greater weight loss in trials
  • Both require gradual dose titration to minimize GI side effects
  • Semaglutide has years of real-world safety data
  • Retatrutide may have different side effect profile due to glucagon activation
  • Cost and availability strongly favor Semaglutide currently
  • Retatrutide's glucagon component may better preserve muscle during weight loss

Melanotan II vs PT-141 (Bremelanotide)

Melanotan II

Non-Selective Melanocortin Receptor Agonist

Description

Synthetic peptide analog of alpha-melanocyte stimulating hormone (α-MSH). Melanotan II activates multiple melanocortin receptors (MC1R, MC3R, MC4R, MC5R), producing tanning, appetite suppression, and sexual effects.

Mechanism of Action

Non-selective activation of melanocortin receptors throughout the body. MC1R activation produces melanogenesis (tanning), MC3R/MC4R activation suppresses appetite and enhances sexual arousal, MC5R affects sebaceous glands. The broad receptor activation creates multiple effects simultaneously.

Key Benefits

  • UV-independent skin tanning and pigmentation
  • Enhanced sexual arousal and libido (both sexes)
  • Appetite suppression and potential weight loss
  • Possible photoprotective effects
  • Increased skin pigmentation provides some UV protection
  • Effects on multiple systems simultaneously
Dosing: Subcutaneous injection: Loading phase of 0.25-0.5mg daily until desired tan achieved, then maintenance of 0.25-1mg 2-3 times weekly
Duration: Half-life of approximately 1 hour, but melanogenesis effects accumulate over days to weeks
VS

PT-141 (Bremelanotide)

Selective Melanocortin Receptor Agonist (MC3R/MC4R)

Description

Selective melanocortin receptor agonist developed specifically for sexual dysfunction. PT-141 is a cyclic metabolite of Melanotan II that was FDA-approved as Vyleesi for female hypoactive sexual desire disorder.

Mechanism of Action

Selective activation of MC3R and MC4R receptors in the central nervous system, particularly the hypothalamus. Enhances sexual desire through CNS-mediated pathways rather than vascular mechanisms (unlike Viagra). Does not activate MC1R, so produces no tanning effect.

Key Benefits

  • Powerful enhancement of sexual desire and arousal
  • Effective in both males and females
  • Works through CNS mechanisms (not vascular)
  • FDA-approved for female sexual dysfunction
  • No tanning or pigmentation effects
  • May improve erectile function through desire pathways
  • Rapid onset of effects (within 1-2 hours)
Dosing: Subcutaneous injection: 1-2mg administered 30-45 minutes before anticipated sexual activity. FDA-approved dosing for females is 1.75mg.
Duration: Short half-life but effects on arousal last 6-12 hours

The Verdict

Melanotan II is ideal for subjects seeking tanning effects along with sexual enhancement and appetite suppression - a multi-purpose peptide. PT-141 is superior for pure sexual enhancement without tanning, offering more potent and selective effects on libido and arousal. PT-141 is FDA-approved for sexual dysfunction and provides more predictable sexual effects. Choose Melanotan II for tanning + sexual benefits; choose PT-141 for maximum sexual enhancement without pigmentation.

Key Considerations

  • Melanotan II causes tanning; PT-141 does not
  • PT-141 is FDA-approved for sexual dysfunction; Melanotan II is not
  • PT-141 provides more potent sexual effects without other effects
  • Melanotan II offers multiple benefits (tanning, appetite, libido)
  • Both can cause nausea, especially at higher doses
  • PT-141 is better for on-demand use before sexual activity
  • Melanotan II requires loading phase for tanning; PT-141 works acutely
  • Melanotan II may cause darkening of existing moles and freckles

Semax vs Selank

Semax

Synthetic ACTH Analog (Nootropic Peptide)

Description

Russian-developed synthetic peptide derived from ACTH (adrenocorticotropic hormone). Semax is a heptapeptide with powerful nootropic, neuroprotective, and cognitive-enhancing properties.

Mechanism of Action

Increases BDNF (brain-derived neurotrophic factor) expression, enhances neuroplasticity, modulates dopamine and serotonin systems, and provides neuroprotection through multiple pathways. Improves cerebral blood flow and oxygen utilization. Enhances attention and memory formation.

Key Benefits

  • Enhanced cognitive function and mental clarity
  • Improved memory formation and recall
  • Increased focus and concentration
  • Neuroprotective effects
  • Enhanced learning capacity
  • Improved stress resilience
  • Potential benefits for ADHD-like symptoms
  • Increased mental energy without stimulation
Dosing: Intranasal administration: 2-3 drops (400-600mcg) per nostril, 1-2 times daily. Higher concentration versions (0.1%, 1%) available for enhanced effects.
Duration: Short half-life but neuroplastic effects accumulate with consistent use
VS

Selank

Synthetic Tuftsin Analog (Anxiolytic Peptide)

Description

Russian-developed synthetic peptide derived from the immunomodulatory peptide tuftsin. Selank provides anxiolytic effects with cognitive enhancement, representing a unique non-sedating anti-anxiety agent.

Mechanism of Action

Modulates GABA, serotonin, and dopamine systems without direct receptor binding. Increases BDNF, enhances enkephalin metabolism, and stabilizes emotional regulation. Provides anxiolytic effects while maintaining or enhancing cognitive function through multiple neurotransmitter pathways.

Key Benefits

  • Anxiolytic effects without sedation
  • Enhanced cognitive function
  • Improved stress resilience and emotional stability
  • Better focus under stress
  • No tolerance or dependence
  • Immune system modulation
  • Improved sleep quality
  • Neuroprotective properties
Dosing: Intranasal administration: 2-3 drops (400-600mcg) per nostril, 2-3 times daily. Can be used as needed for acute stress or daily for baseline anxiety.
Duration: Short half-life but effects on neurotransmitter systems persist throughout the day

The Verdict

Semax is superior for pure cognitive enhancement, focus, memory, and learning - ideal for demanding mental work or studying. Selank excels at anxiety reduction and stress management while maintaining cognitive function - perfect for high-stress situations or chronic anxiety. Many researchers use both together: Semax for cognitive boost and Selank for anxiety control, creating a synergistic nootropic and anxiolytic combination.

Key Considerations

  • Semax is better for cognitive enhancement and focus
  • Selank is superior for anxiety and stress management
  • Both enhance BDNF and provide neuroprotection
  • Semax may be too stimulating for anxious individuals
  • Selank is better for sleep quality
  • Both can be used together synergistically
  • Neither causes dependence or tolerance
  • Both are administered intranasally for convenience

⚠️ Research Use Only

All comparisons are provided for educational and research purposes only. These substances are intended for laboratory research and are not for human consumption. Consult qualified healthcare professionals and ensure compliance with all applicable regulations in your jurisdiction. The information provided represents research findings and does not constitute medical advice.